Posted by: jerseyg | September 20, 2009

The Creation of the 2009 Flu Pandemic out of the Ashes of the Deadly 1918 Pandemic-It’s All in the Digging…..


by Robert S. Finnegan

Link to original article here

In 1997, freelance investigative reporter Robert S. Finnegan, then living and based in Alaska was present when Dr. John Hultin managed to con the Natives of Brevig Mission Alaska into allowing him to exhume the bodies of several Native Alaskans against the almost violent protests of the Natives. Plying the Natives with lies and money, Hultin managed to find two corpses that appeared still viable for extraction of the H5N1 DNA buried in the permafrost. One corpse retained the viral DNA in its desiccated lungs. He then returned to the lower 48 with enough DNA to resurrect this killer virus that had been eradicated in the early 1900’s. Hultin turned virus samples over to the CDC and the U.S. Army (Jeffery Taubenberger) who then proceeded to weaponize the virus H5N1, baptizing his new doomsday creation with the name “Bird Flu.” This virus that had long been eradicated has now been released once more to kill again, this time possibly billions of human beings across the planet, by combining with the brand new killer H1N1 that has the capability to cross barriers in pigs, birds and humans becoming a “Super-Super” virus.

Today, on this date the world is seeing the results of this evil “research” as a new pandemic roars out of Mexico into the United States. I was in Alaska when this travesty occurred. Unfortunately, the Alaska Native Federation was unable to stop this theft. “Doctor” Hulin, the CDC, and the United States Army are guilty of crimes against humanity – especially the CDC – who are now in the business of killing rather than curing, and probably have been for some time now under the Bush/Cheney crime regime. The blame for this pandemic, if it indeed becomes one, can be laid squarely at their feet. I have been waiting for this to occur since 1997. Martial Law, forced vaccinations and mass death will follow shortly if this comes to pass.

Perhaps we slept too long.

Origin of Virus

One theory is that the virus strain originated at Fort Riley, Kansas, by two genetic mechanisms — genetic drift and antigenic shift — in viruses in poultry and swine which the fort bred for local consumption. Though initial data from a recent reconstruction of the virus suggested that it jumped directly from birds to humans, without traveling through swine [1], this has since been cast into doubt. One researcher argues that the disease was found in Haskell County, Kansas as early as January 1918. [2]

Discovery of viral genomes

In February 1997, Johan Hultin recovered samples of the 1918 influenza from the frozen corpse of a Native Alaskan woman buried for nearly eight decades in permafrost near Brevig Mission, Alaska [3]. He brought the samples to a team led by Jeffery Taubenberger of the US Armed Forces Institute of Pathology (AFIP). Brevig Mission lost approximately 85% of its population to the 1918 flu in November 1918. One of the four recovered samples contained viable genetic material of the virus. This sample provided scientists a first-hand opportunity to study the virus, which was inactivated with guanidinium thiocyanate before transport. This sample and others found in AFIP archives allowed researchers to completely analyze the critical gene structures of the 1918 virus. “We have now identified three cases: the Brevig Mission case and two archival cases that represent the only known sources of genetic material of the 1918 influenza virus”, said Taubenberger, chief of AFIP’s molecular pathology division and principal investigator on the project. [4] [5]

The February 6, 2004 edition of Science magazine reported that two research teams, one led by Sir John Skehel, director of the National Institute for Medical Research in London, another by Professor Ian Wilson of The Scripps Research Institute in San Diego, had managed to synthesize the hemagglutinin protein responsible for the 1918 flu outbreak of 1918. They did this by piecing together DNA from a lung sample from an Inuit woman buried in the Alaskan tundra and a number of preserved samples from American soldiers of the First World War. The teams had analyzed the structure of the gene and discovered how subtle alterations to the shape of a protein molecule had allowed it to move from birds to humans with such devastating effects.

On October 5, 2005, researchers at the Mount Sinai School of Medicine in New York announced that the genetic sequence of the 1918 flu strain, a subtype of avian strain H1N1, had been reconstructed using historic tissue samples. [6] [7] [8]

Characteristics of virus

Influenza viruses have a relatively high mutation rate that is characteristic of RNA viruses. The H5N1 virus has mutated into a variety of types with differing pathogenic profiles; some pathogenic to one species but not others, some pathogenic to multiple species. [9] The ability of various influenza strains to show species-selectivity is largely due to variation in the hemagglutinin genes. Genetic mutations in the hemagglutinin gene that cause single amino acid substitutions can significantly alter the ability of viral hemagglutinin proteins to bind to receptors on the surface of host cells. Such mutations in avian H5N1 viruses can change virus strains from being inefficient at infecting human cells to being as efficient in causing human infections as more common human influenza virus types. [10] This doesn’t mean one amino acid substitution can cause a pandemic but it does mean one amino acid substitution can cause an avian flu virus that is not pathogenic in humans to become pathogenic in humans.

In July 2004, researchers led by H. Deng of the Harbin Veterinary Research Institute, Harbin, China and Robert Webster of the St Jude Children’s Research Hospital, Memphis, Tennessee, reported results of experiments in which mice had been exposed to 21 isolates of confirmed H5N1 strains obtained from ducks in China between 1999 and 2002. They found “a clear temporal pattern of progressively increasing pathogenicity”. [11] Results reported by Webster in July 2005 reveal further progression toward pathogenicity in mice and longer virus shedding by ducks.

In December, 2008, research by Yoshihiro Kawaoka of University of Wisconsin linked the presence of the three specific genes (termed PA, PB1, and PB2) and a nucleoprotein derived from the 1918 flu samples was enough to trigger similar symptoms in animal testing. [12]

Research of viral pathogenesis

Recent research of Taubenberger et al has suggested that the 1918 virus, like H5N1, could have arisen directly from an avian influenza virus. [13] However, researchers at University of Virginia and Australian National University have suggested that there may be an alternative interpretation of the data used in the Taubenberger et al. paper. [14] [15] Taubenberger et al responded to these letters and defended their original interpretation. [16]

Other research by Tumpey and colleagues who reconstructed the H1N1 virus of 1918 came to the conclusion that it is was most notably the polymerase genes and the HA and NA genes that caused the extreme virulence of this virus. [17] The sequences of the polymerase proteins (PA, PB1, and PB2) of the 1918 virus and subsequent human viruses differ by only 10 amino acids from the avian influenza viruses. Viruses with seven of the ten amino acids in the human influenza locations have already been identified in currently circulating H5N1. This has led some researchers to suggest that other mutations may surface and make the H5N1 virus capable of human-to-human transmission. Another important factor is the change of the HA protein to a binding preference for alpha 2,6 sialic acid (the major form in the human respiratory tract). In avian virus the HA protein preferentially binds to alpha 2,3 sialic acid, which is the major form in the avian enteric tract. It has been shown that only a single amino acid change can result in the change of this binding preference. Altogether, only a handful of mutations may need to take place in order for H5N1 avian flu to become a pandemic virus like the one of 1918. However it is important to note that likelihood of mutation does not indicate the likelihood for the evolution of such a strain; since some of the necessary mutations may be constrained by stabilizing selection.

On 18 January 2007, Kobasa et al reported that infected monkeys ( Macaca fascicularis) exhibited classic symptoms of the 1918 pandemic and died from a cytokine storm. [18]

Blood plasma as an effective treatment

In the event of another pandemic, US military researchers have proposed reusing a treatment from the deadly pandemic of 1918 in order to blunt the effects of the flu. Some military doctors injected severely afflicted patients with blood or blood plasma from people who had recovered from the flu. Data collected during that time indicates that the blood-injection treatment reduced mortality rates by as much as 50 percent. Navy researchers have launched a test to see if the 1918 treatment will work against deadly Asian bird flu. Results thus far have been inconclusive. Human H5N1 plasma may be an effective, timely, and widely available treatment for the next flu pandemic. A new international study using modern data collection methods, would be a difficult, slow process. But many flu experts, citing the months-long wait for a vaccine for the next pandemic, are of the opinion that the 1918 method is something to consider. [19]

In the world wide 1918 flu pandemic, “physicians tried everything they knew, everything they had ever heard of, from the ancient art of bleeding patients, to administering oxygen, to developing new vaccines and sera (chiefly against what we now call Hemophilus influenzae—a name derived from the fact that it was originally considered the etiological agent—and several types of pneumococci). Only one therapeutic measure, transfusing blood from recovered patients to new victims, showed any hint of success.” [20]

Sources and notes

1. Sometimes a virus contains both avian adapted genes and human adapted genes. Both the H2N2 and H3N2 pandemic strains contained avian flu virus RNA segments. “While the pandemic human influenza viruses of 1957 (H2N2) and 1968 (H3N2) clearly arose through reassortment between human and avian viruses, the influenza virus causing the ‘ 1918 flu‘ in 1918 appears to be entirely derived from an avian source (Belshe 2005).” (from Chapter Two : Avian Influenza by Timm C. Harder and Ortrud Werner, an excellent free on-line Book called Influenza Report 2006 which is a medical textbook that provides a comprehensive overview of epidemic and pandemic influenza.)
2. The site of origin of the 1918 influenza pandemic and its public health implications
3. Washington Post article
4. Lethal secrets of 1918 flu virus; BBC
5. According to Gina Kolata’s 1999 account of the pandemic, ‘Flu’, pp. 255-65 : Johan Hultin first attempted to recover samples from Brevig in 1951, but he was unsuccessful. In 1997, by then a seventy-two year old retired pathologist, he decided that science had advanced enough to make another attempt worthwhile. Taubenberger had already recovered RNA of limited quality from samples of two servicemen who had died in the pandemic, and Hultin wrote offering offering his services to try to get better quality samples from Brevig permafrost. Taubenberger accepted, and Hultin went alone to Brevig in August 1997, and recovered the sample from the Alaskan woman, which Taubenberger and his team then analysed.
6. Special report at Nature News: The 1918 flu virus is resurrected, Published online: 5 October 2005; doi:10.1038/437794a
7. Taubenberger, Jeffery K.; Ann H. Reid, Raina M. Lourens, Ruixue Wang, Guozhong Jin and Thomas G. Fanning (2005). “Characterization of the 1918 influenza virus polymerase genes”. Nature 437: 889–893. doi:10.1038/nature04230.
8. Also: Tumpey, Terrence M.; Christopher F. Basler, Patricia V. Aguilar, Hui Zeng, Alicia Solórzano, David E. Swayne, Nancy J. Cox, Jacqueline M. Katz, Jeffery K. Taubenberger, Peter Palese and Adolfo García-Sastre (2005). “Characterization of the Reconstructed 1918 Spanish Influenza Pandemic Virus”. Science 310: 77–80. doi:10.1126/science.1119392. PMID 16210530.
9. Kou, Z.; F. M. Lei, J. Yu, Z. J. Fan, Z. H. Yin, C. X. Jia, K. J. Xiong, Y. H. Sun, X. W. Zhang, X. M. Wu, X. B. Gao and T. X. Li (2005). “New genotype of avian influenza H5N1 viruses isolated from tree sparrows in China”. Journal of Virology 79: 15460–15466. doi:10.1128/JVI.79.24.15460-15466.2005. PMID 16306617.
10. Evolution of the receptor binding phenotype of influenza A (H5) viruses by A. Gambaryan, A. Tuzikov, G. Pazynina, N. Bovin, A. Balish and A. Klimov in Virology (2005) electronic release on October 11 ahead of print publication.
11. The evolution of H5N1 influenza viruses in ducks in southern China by H. Chen, G. Deng, Z. Li, G. Tian, Y. Li, P. Jiao, L. Zhang, Z. Liu, R. G. Webster and K. Yu in Proceedings of the National Academy of Sciences of the United States of America (2004) volume 101, pages 10452-10457.
12. Reuters
13. Recent research of Taubenberger et al has suggested that the 1918 virus, like H5N1, could have arisen directly from an avian influenza virus in: Taubenberger JK, Reid AH, Lourens RM, Wang R, Jin G, Fanning TG. Characterization of the 1918 influenza virus polymerase genes. Nature. October 6, 2005;437(7060):889-893
14. Was the 1918 pandemic caused by a bird flu? – Gibbs and Gibbs Nature. April 27, 2006;440:E8
15.Was the 1918 flu avian in origin? – Antonovics et al. Nature. April 27, 2006;440:E9
16. Molecular virology: Was the 1918 pandemic caused by a bird flu? Was the 1918 flu avian in origin? (Reply)
17. Tumpey TM, Basler CF, Aguilar PV, Zeng H, Solorzano A, Swayne DE, Cox NJ, Katz JM, Taubenberger JK, Palese P, Garcia-Sastre A. Characterization of the reconstructed 1918 Spanish influenza pandemic virus. Science. October 7, 2005;310(5745):77-80
18. Aberrant innate immune response in lethal infection of macaques with the 1918 influenza virus Nature. 18 January 2007;445:319
20. The Threat of Pandemic Influenza: Are We Ready? Workshop Summary (2005) (free online book) page 62
Retrieved from “
By Robert S. Finnegan



  1. I have a Sep. 5, 2009 report by board-certified neurosurgeon, Dr. Russell Blaylock, saying that there is no evidence worldwide that H1N1 has mutated. No one’s saying it can’t; it just hasn’t done so yet. H1N1 is very mild, and historical data now suggests that the 1918 flu actually killed far fewer people than what corpo-gov tells us. In fact, he says that many people died of pneumonia, not flu, back then.

    No one called it a flu pandemic in 1918 – no one knew they were all dealing with the same bug. Records weren’t amassed till decades later – and from that collection comes our current “historic” view of what happened in 1918.

    Blaylock says there are two sets of info about H1N1 – that which comes from corpo-gov and that which comes from doctors. Contrary to MSM reports, there is no “progressively increasing pathogenicity” for H1N1, and a very low infectivity rate.

    Finnegan raises the cytokine storm, which the fearmongers are using to explain why 20 year olds are more at risk of dying (tho, the death rate from H1N1 is at 0.03%, so of little concern, and less lethal than the last two years’ flu variants, per Blaylock):

    “On 18 January 2007, Kobasa et al reported that infected monkeys … exhibited classic symptoms of the 1918 pandemic and died from a cytokine storm.”

    In video 4, Blaylock details many homeopathic techniques that will dampen cytokine storming, including increasing your D3 intake to 10,000 International Units a day (2,000 for kids), and supplementing your selenium intake. A cold compress on your fever for the first several days helps tremendously. (On again, off again, so as not to damage your skin.)

    It’s good to keep track of what the sociopaths have been up to, and what can protect you and yours.

    • Hello Rady,

      Thank you for your comments. When I took this investigation on I knew I was going to have problems without experts to set straight any errors I made. Fortunately it attracted the attention of a microbiologist actually working on the recombination and she walked me through it. There was no doubt in her mind that this “novel” virus was man-made and could not have mutated in the wild in less than 100 years. The only reason I was involved was the fact that I resided in Alaska at the time, and was friends with the Native Americans there. This should never have happened to them ad I wanted to see justice done as they had no other voice at the time.

      What do you think about this just disappearing into thin air after all the falsehoods and scare tactics generated by the White House and England? As I stated I know nothing about microbiology and recombination but it looks to me as though they intended to “cull the herd,” and blew it. We dodged a bullet on this one, and this after they basically came out and said they were going to release it. Too many loose ends on this. They are destroying some of the vaccine right now. It would be interesting to get an independent analysis and breakdown of what exactly is in that stew.

      Thanks also to Dr. Shen Tenpenny.

      Robert S. Finnegan
      Southeast Asia Independent Media
      Jakarta, Indonesia

  2. Actually, Dr Sheri Tenpenny states that the three staged vaccines they are planning to deliver may create the “perfect cytokine storm”..

    Pdf file

    I also don’t doubt for a minute that the numbers have been exaggerated by those with ulterior motives and honestly misdiagnosed by doctors to boot. I’m sure many that died from the flu already had compromised immune systems. Plus, one has to wonder just how good their record keeping was back in the day.

  3. yeah, and I have no basis to doubt that the 3-series vaccine can cause a cytokine storm…

    Blaylock is merely offering techniques to dampen the storm, if you HAVE TO take the shots.

  4. Yea, we posted his “what to do if you are forced to take the swine flu shot” advice a while back.

    I have no intention of getting a shot but I printed it out and stashed it in my desk….. just in case……….


    Coloidal Silver

    D3 FOODS
    Grain fed yolks


    Green or Red peppers (nightshade)


    Apple Cider Vinegar
    Lemon Oil
    apple or citrus pectin

    (Oxygen Radical Absorbance Capacity)
    Goji Berries
    Dark Chocolate
    Black Raspberries
    Milk Chocolate
    Red Raspberries
    Yellow Squash
    Brussels Sprouts
    Alfalfa Sprouts
    Red Grapes
    Red Bell Pepper
    Baked Beans
    White Grapefruit
    Kidney Beans
    White Grapes
    Frozen Peas
    Sweet Potato
    Cucumber 739

    NUDE SUNBATHING – 20-30 minutes (my favorite)


    • Is that the recipe from your grandpa, Patrick?

      • Yes Curt, We called him Daddo, the FDA called him a quack and the neighbors called him a naked nuicance :0

    • Mmmmm chocolate and raspberrries goes so well together…….but I thought only dark chocolate was beneficial. I personally prefer the sweet creamy taste of milk chocolate so I’m glad it made the list.

      • All Chocolate is good. Not the sugar, but in this case the milk does have some benefit. Daddo would say drop the chocolate. Also important to note that these
        vegetables need to eaten uncooked or flash steamed.

        Cooking kills, literally. Eating raw eggs and sushi was fine until the agri-terrorists got involved with Henry Ford’s Factory essembly line mentalities.

        Daddo’s Tonic:

        Alfalfa/peppermint Tea – 8oz.
        teaspoon of CiderVinegar/Pectin
        tablespoon Raw unfiltered honey

        Delicious, even as a four year old boy

  6. Purrrrrrrrrrrrrr …………

    Catnip can also be smoked recreationally, and when combined with tobacco and other herbs, provides a “minty” taste and mild intoxicating effects

  7. Sounds yummy pod. As long as the cats don’t light up in the house 😉

  8. Garden naked…make the National Guard burn alot of fuel hovering at your site.

  9. btw – did you see the klown again lied about why he banned everyone? must be feeling terribly insecure

    he put it in one of his nervous nelly emails about having been hacked

  10. He what?! Do tell. I am not privy to the klown man’s emails. What’s he sayin’, he banned us all because he was hacked???

  11. Hello Rady Darlin,

    So Robby lied, again? Hmmm! Traffic on the site has been down. Then again OEN photographer Cheryl Biren was arrested last week at the Army Experience for taking pictures during the protest. That made some news.

    I know when I visit the site I get the hourglass. If ya know what I mean?

    Nothing really there. OEN has lost it’s mojo.

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